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IJMS | Free Full-Text | Alleviation of LPS-Induced Inflammation and Septic  Shock by Lactiplantibacillus plantarum K8 Lysates
IJMS | Free Full-Text | Alleviation of LPS-Induced Inflammation and Septic Shock by Lactiplantibacillus plantarum K8 Lysates

Lipopolysaccharide (LPS)-Induced Biliary Epithelial Cell NRas Activation  Requires Epidermal Growth Factor Receptor (EGFR) | PLOS ONE
Lipopolysaccharide (LPS)-Induced Biliary Epithelial Cell NRas Activation Requires Epidermal Growth Factor Receptor (EGFR) | PLOS ONE

Inhibition of Lipopolysaccharide-Induced Inflammatory Responses by an  Apolipoprotein AI Mimetic Peptide | Circulation Research
Inhibition of Lipopolysaccharide-Induced Inflammatory Responses by an Apolipoprotein AI Mimetic Peptide | Circulation Research

Surfactant Protein A Enhances the Degradation of LPS-Induced TLR4 in  Primary Alveolar Macrophages Involving Rab7, β-arrestin2, and mTORC1 |  Infection and Immunity
Surfactant Protein A Enhances the Degradation of LPS-Induced TLR4 in Primary Alveolar Macrophages Involving Rab7, β-arrestin2, and mTORC1 | Infection and Immunity

LPS‑induced proinflammatory cytokine expression in human airway epithelial  cells and macrophages via NF‑κB, STAT3 or AP‑1 activation
LPS‑induced proinflammatory cytokine expression in human airway epithelial cells and macrophages via NF‑κB, STAT3 or AP‑1 activation

Aspirin inhibits LPS-induced macrophage activation via the NF-κB pathway |  Scientific Reports
Aspirin inhibits LPS-induced macrophage activation via the NF-κB pathway | Scientific Reports

Azilsartan Suppressed LPS-Induced Inflammation in U937 Macrophages through  Suppressing Oxidative Stress and Inhibiting the TLR2/MyD88 Signal Pathway |  ACS Omega
Azilsartan Suppressed LPS-Induced Inflammation in U937 Macrophages through Suppressing Oxidative Stress and Inhibiting the TLR2/MyD88 Signal Pathway | ACS Omega

Lipopolysaccharides induced inflammatory responses and electrophysiological  dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes  | Scientific Reports
Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes | Scientific Reports

The LPS-Induced Transcriptional Upregulation of the Chicken Lysozyme Locus  Involves CTCF Eviction and Noncoding RNA Transcription: Molecular Cell
The LPS-Induced Transcriptional Upregulation of the Chicken Lysozyme Locus Involves CTCF Eviction and Noncoding RNA Transcription: Molecular Cell

Lipopolysaccharides induced inflammatory responses and electrophysiological  dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes  | Scientific Reports
Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes | Scientific Reports

Ginsenoside Rg1 attenuates LPS-induced cognitive impairments and  neuroinflammation by inhibiting NOX2 and Ca2+–CN–NFAT1 signaling in mice -  ScienceDirect
Ginsenoside Rg1 attenuates LPS-induced cognitive impairments and neuroinflammation by inhibiting NOX2 and Ca2+–CN–NFAT1 signaling in mice - ScienceDirect

Neuroinflammation induced by lipopolysaccharide causes cognitive impairment  in mice | Scientific Reports
Neuroinflammation induced by lipopolysaccharide causes cognitive impairment in mice | Scientific Reports

Effect of LPS treatment on cell number, DNA fragmentation, and... |  Download Scientific Diagram
Effect of LPS treatment on cell number, DNA fragmentation, and... | Download Scientific Diagram

Dynamin‐related Irgm proteins modulate LPS‐induced caspase‐11 activation  and septic shock | EMBO reports
Dynamin‐related Irgm proteins modulate LPS‐induced caspase‐11 activation and septic shock | EMBO reports

Frontiers | A Novel Lipopolysaccharide Recognition Mechanism Mediated by  Internalization in Teleost Macrophages
Frontiers | A Novel Lipopolysaccharide Recognition Mechanism Mediated by Internalization in Teleost Macrophages

LPS Model of Sepsis | Melior Discovery
LPS Model of Sepsis | Melior Discovery

Lipopolysaccharide-Induced Microglial Activation and Neuroprotection  against Experimental Brain Injury Is Independent of Hematogenous TLR4 |  Journal of Neuroscience
Lipopolysaccharide-Induced Microglial Activation and Neuroprotection against Experimental Brain Injury Is Independent of Hematogenous TLR4 | Journal of Neuroscience

Surfactant Protein A Enhances the Degradation of LPS-Induced TLR4 in  Primary Alveolar Macrophages Involving Rab7, β-arrestin2, and mTORC1 |  Infection and Immunity
Surfactant Protein A Enhances the Degradation of LPS-Induced TLR4 in Primary Alveolar Macrophages Involving Rab7, β-arrestin2, and mTORC1 | Infection and Immunity

SRV2 promotes mitochondrial fission and Mst1-Drp1 signaling in LPS-induced  septic cardiomyopathy | Aging
SRV2 promotes mitochondrial fission and Mst1-Drp1 signaling in LPS-induced septic cardiomyopathy | Aging

A unique hybrid characteristic having both pro- and anti-inflammatory  phenotype transformed by repetitive low-dose lipopolysaccharide in C8-B4  microglia | Scientific Reports
A unique hybrid characteristic having both pro- and anti-inflammatory phenotype transformed by repetitive low-dose lipopolysaccharide in C8-B4 microglia | Scientific Reports

Life | Free Full-Text | Natural Sulfurs Inhibit LPS-Induced Inflammatory  Responses through NF-κB Signaling in CCD-986Sk Skin Fibroblasts
Life | Free Full-Text | Natural Sulfurs Inhibit LPS-Induced Inflammatory Responses through NF-κB Signaling in CCD-986Sk Skin Fibroblasts

LPS‑induced proinflammatory cytokine expression in human airway epithelial  cells and macrophages via NF‑κB, STAT3 or AP‑1 activation
LPS‑induced proinflammatory cytokine expression in human airway epithelial cells and macrophages via NF‑κB, STAT3 or AP‑1 activation

LPS‑induced proinflammatory cytokine expression in human airway epithelial  cells and macrophages via NF‑κB, STAT3 or AP‑1 activation
LPS‑induced proinflammatory cytokine expression in human airway epithelial cells and macrophages via NF‑κB, STAT3 or AP‑1 activation

Potentiation of LPS-Induced Apoptotic Cell Death in Human Hepatoma HepG2  Cells by Aspirin via ROS and Mitochondrial Dysfunction: Protection by  N-Acetyl Cysteine | PLOS ONE
Potentiation of LPS-Induced Apoptotic Cell Death in Human Hepatoma HepG2 Cells by Aspirin via ROS and Mitochondrial Dysfunction: Protection by N-Acetyl Cysteine | PLOS ONE